Why Weight Loss Stops On LONG-TERM Low Carb DietsOn by
I’m confident weight loss stalling not better explained by poor diet is usually related to comparative/absolute leptin insufficiency. The reason I understand this is so is basically because leptin replacement has significantly improved my energy (that have been probably impaired by low free t3 secondary to extreme weight-loss and LC eating). Weight food and loss limitation related thyroid problems shouldn’t be with symptomatic goiter; typical presentation is going to be low TSH with low free T3 and low or normal T4. It is hypothalamic, functional.
Goiter will show a problem with the thyroid itself, either deficient substrate or dysfunction of the gland (and the goiter, hypertrophy, is an indicator of this). In weight/food lower thyroidism, the gland would go to sleep and conversion of the inactive thyroid hormone is silenced by decreased sympathetic anxious system output (supplementary to low leptin, low insulin, low fatty acids, and blood sugar in the bloodstream).
Anecdotally, the reason why I know it is a leptin thing and not a minimal-carb thing is that the energy went skyward after getting leptin even though I still eat LC. If it were my diet of 40-60 carbs daily, why do I’ve energy for days… but easily stop taking leptin, eventually I become a ravenous beast with pitiful energy. All I can say is, insulin might rule fattening, but leptin guidelines weight maintenance. If medicine were as smart as me, it would start screening and treating this substantial reduction in leptin which occurs after weight dieting and reduction.
- Time-efficient exercises to improve your metabolism
- I combine in HIIT (High Intensity INTENSIVE TRAINING)
- 1/2 cup Marinara Sauce
- You cannot achieve a sound body weight, even through medically supervised dieting
At least in a specific portion of the dieting/obese people (people who KNOW what works and WANT to do it) this treatment is the difference between being miserable or excess fat as being both happy content AND thin/energetic. They know successful weight losers have extreme and sharpened drops in leptin. For some good reason they think this is an excellent thing.
Somehow leptin, in weight problems research, became bad. It’s crazy. It started with the observation that fatty unwanted fat fats have high leptin, so when they lose weight, the leptin boils down (tendencies toward decreasing). So somehow leptin being lower after weight loss became to be interpreted as a very important thing, just like insulin decreases and glucose lowers. Sure elevated leptin is an indicator of metabolic disease, and symptomatic fat cell hypertrophy. Sure high leptin can cause problems when it is at the known levels within the obese and hyperinsulinemic.
That doesn’t indicate “low leptin is good” after weight reduction! I mean, visualize if people in respiratory distress were known to manifest increased respirations; quality of respiratory distress will correlate with a decrease in respiratory system rate and work, right? So it is good then that people have respiratory rates of 7/min? That is so when the topic is leptin. Stop complicated tendencies with causes. Reduction in a value might indicate improvement, but sometimes it also may be the body’s attempt at stopping what it perceives to be an imbalance. That attempt works well.
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